CDK2/cyclinA inhibitors: targeting the cyclinA recruitment site with small molecules derived from peptide leads

Bioorg Med Chem Lett. 2006 Mar 15;16(6):1716-20. doi: 10.1016/j.bmcl.2005.12.004. Epub 2005 Dec 27.

Abstract

The syntheses of potent small molecule inhibitors of the CDK2/cyclinA recruitment site are described. Structure-activity trends of nanomolar octapeptides were examined through amino-acid substitution and truncation of the sequence resulting in the identification of a smaller, albeit significantly less potent, tetrapeptide lead. These losses in affinity were recovered by side-chain optimization and by rigidification of the peptide backbone using a combination of solid-phase parallel synthesis and structure-based design. Finally, two guanidine functionalities were replaced to improve drug-like properties, resulting in neutral small molecules equal in activity to that of the peptide lead.

MeSH terms

  • Amino Acid Substitution
  • Cyclin A / antagonists & inhibitors*
  • Cyclin A / chemistry
  • Cyclin-Dependent Kinase 2 / antagonists & inhibitors*
  • Cyclin-Dependent Kinase 2 / chemistry
  • Drug Design
  • Enzyme Inhibitors / chemistry
  • Enzyme Inhibitors / pharmacology*
  • Guanidine / chemistry
  • Humans
  • Models, Molecular
  • Peptide Fragments / chemistry
  • Peptide Fragments / pharmacology*
  • Protein Conformation

Substances

  • Cyclin A
  • Enzyme Inhibitors
  • Peptide Fragments
  • Cyclin-Dependent Kinase 2
  • Guanidine