Vascular endothelial growth factor inhibits programmed cell death of endothelial cells induced by clinorotation

J Gravit Physiol. 2004 Jul;11(2):P199-200.

Abstract

The principal aim of this study was to investigate short- and long-term effects of clinorotation on human endothelial cells (EA hy 926 cell line) using a three-dimensional random positioning machine. Moreover, the impact of vascular endothelial growth factor (VEGF) was addressed. Immediately, within one hour and after four and twenty-four hours an increase of apoptotic cells was detected. VEGF significantly inhibited the amount of apoptotic endothelial cells (EC). VEGF reduced the amount of fas-positive EC. Moreover, after 24 hours, proliferating EC grew in form of three-dimensional multicellular spheroids and also as monolayers. The initially formed spheroids (maximum diameter 3 mm) remained stable up to the 15th day of clinorotation. Some spheroids revealed tubular structures. In addition, a clear increase of extracellular matrix proteins such as osteopontin and fibronectin was measured. The three-dimensional clinostat represents an important tool for cell biological experiments. VEGF significantly attenuated the changes of endothelial cells induced by simulated weightlessness in a cell protective manner.

MeSH terms

  • Apoptosis / drug effects
  • Endothelial Cells / drug effects*
  • Endothelial Cells / physiology*
  • Endothelial Cells / ultrastructure
  • Extracellular Matrix Proteins / physiology
  • Flow Cytometry
  • Humans
  • Microscopy, Fluorescence
  • Rotation
  • Vascular Endothelial Growth Factor A / pharmacology*
  • Weightlessness Simulation*

Substances

  • Extracellular Matrix Proteins
  • Vascular Endothelial Growth Factor A