HIV-1 pol replication capacity predicts disease progression

AIDS. 2005 Jun 10;19(9):871-7. doi: 10.1097/01.aids.0000171400.15619.e1.

Abstract

Objective: To determine the influence of pol replication capacity on the natural history of HIV-1 infection.

Design: Pol replication capacity was measured using a recombinant virus single cycle assay on baseline plasma specimens from subjects enrolled in the Hemophilia Growth and Development Study.

Setting: Children and adolescents with hemophilia and HIV-1 infection were enrolled at multiple sites across the USA into a natural history study.

Participants: The Hemophilia Growth and Development Study enrolled 207 HIV-1-infected hemophiliacs between 6 and 19 years of age in 1989 and 1990. Subjects were followed every 6 months through 1997 with pol replication capacity measurements available from 128 of the subjects.

Main outcome measures: A univariate model defined the relationship between pol replication capacity and HIV-1 RNA and CD4 T-cell number. A random effects model assessed the ability of this measure to predict CD4 T-cell decline over time and a Cox proportional hazards model and Kaplan-Meier analyses defined how it predicts clinical progression.

Results: Pol replication capacity measures correlated with baseline HIV-1 RNA, R = 0.189 (P = 0.03) and CD4 T-cell number, -0.197 (P = 0.03). It also independently predicted CD4 T-cell decline over time and progression to AIDS.

Conclusions: This study demonstrates that pol replication capacity independently influences the natural history of HIV-1 infection.

Publication types

  • Multicenter Study
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adolescent
  • Adult
  • CD4 Lymphocyte Count
  • Child
  • Disease Progression
  • Epidemiologic Methods
  • Genes, pol*
  • HIV Infections / immunology
  • HIV Infections / virology*
  • HIV-1 / genetics*
  • HIV-1 / isolation & purification
  • HIV-1 / physiology
  • Hemophilia A / complications
  • Humans
  • Male
  • RNA, Viral / genetics
  • Viral Load
  • Virus Replication

Substances

  • RNA, Viral