Abstract
Metazoan cells have two pathways for intron removal involving the U2- and U12-type spliceosomes, which contain mostly nonoverlapping sets of small nuclear ribonucleoproteins. We show that in vitro splicing of a U12-type intron assembles an exon junction complex (EJC) that is comparably positioned and contains many of the same components as that deposited by the U2-type spliceosome. The presence of a U12-type intron downstream of a premature termination codon within an open reading frame (ORF) induces nonsense-mediated decay of the mRNA in vivo. These findings suggest a common pathway for EJC assembly by the two spliceosomes and highlight the evolutionary age of the EJC and its downstream functions in gene expression.
Publication types
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Research Support, Non-U.S. Gov't
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Research Support, U.S. Gov't, P.H.S.
MeSH terms
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Cell Line
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Cell Nucleus / metabolism
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Codon, Nonsense
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DNA, Complementary / metabolism
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Evolution, Molecular
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Exons
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Gene Expression Regulation
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HeLa Cells
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Humans
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Immunoprecipitation
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Introns*
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Mutagenesis, Site-Directed
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Open Reading Frames
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Plasmids / metabolism
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RNA / chemistry
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RNA / metabolism
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RNA Precursors / metabolism
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RNA Splicing*
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RNA, Messenger / metabolism*
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RNA, Small Nuclear / genetics*
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RNA, Small Nuclear / metabolism
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Ribonuclease H / chemistry
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Ribonucleoproteins, Small Nuclear / metabolism
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Spliceosomes / metabolism
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Time Factors
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Transfection
Substances
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Codon, Nonsense
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DNA, Complementary
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RNA Precursors
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RNA, Messenger
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RNA, Small Nuclear
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Ribonucleoproteins, Small Nuclear
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U12 small nuclear RNA
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RNA
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Ribonuclease H