Developmental changes of Ni(2+) sensitivity and automaticity in Nkx2.5-positive cardiac precursor cells from murine embryonic stem cell

Circ J. 2004 Jul;68(7):724-6. doi: 10.1253/circj.68.724.

Abstract

Background: It is controversial which subtypes of T type Ca(2+) channels are implicated in automaticity of cardiac cells during the embryonic period.

Method and results: The effect of Ni(2+) on the automaticity of Nkx2.5-positive cardiac precursor cells sorted from embryonic stem cells during their differentiation was examined using patch clamp techniques. Although 40 micromol/L Ni(2+), which is enough to block Ni(2+)sensitive T type-Ca(2+) channels, decreased the spontaneous beating rate in all cells in the early and intermediate stage, Ni(2+) did not show any effects on the automaticity of 50% of the cells in the late stage.

Conclusion: These results indicate that Ni(2+)-sensitive T-type Ca(2+) channels expressed in the Nkx2.5-positive cardiac precursor cells are developmentally regulated.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Calcium Channels / drug effects
  • Calcium Channels / physiology
  • Cell Cycle / drug effects
  • Embryonic Development / physiology*
  • Heart / embryology*
  • Homeobox Protein Nkx-2.5
  • Homeodomain Proteins / physiology*
  • Mice
  • Nickel / pharmacology*
  • Protein Precursors / drug effects
  • Protein Precursors / physiology*
  • Stem Cells / cytology*
  • Transcription Factors / physiology*

Substances

  • Calcium Channels
  • Homeobox Protein Nkx-2.5
  • Homeodomain Proteins
  • Nkx2-5 protein, mouse
  • Protein Precursors
  • Transcription Factors
  • Nickel