Abstract
While transfection of tlr2 conveyed responsiveness to lipoteichoic acid (LTA), the Arg753Gln polymorphic gene could not. LTA induced a stronger chemokine and anti-inflammatory response than lipopolysaccharides did. Blood from heterozygous polymorphic and wild-type donors reacted uniformly to LTA and Staphylococcus aureus. Thus, one functional allele for Toll-like receptor 2 suffices for full cytokine response.
MeSH terms
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Chemokines / biosynthesis*
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Heterozygote
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Humans
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In Vitro Techniques
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Inflammation Mediators / metabolism*
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Interferon-gamma / biosynthesis
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Interleukin-10 / biosynthesis
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Interleukin-8 / biosynthesis
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Lipopolysaccharides / pharmacology*
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Membrane Glycoproteins / genetics*
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Mutation, Missense
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Polymorphism, Genetic*
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Receptors, Cell Surface / genetics*
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Teichoic Acids / pharmacology*
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Toll-Like Receptor 2
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Toll-Like Receptors
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Transfection
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Tumor Necrosis Factor-alpha / biosynthesis
Substances
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Chemokines
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Inflammation Mediators
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Interleukin-8
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Lipopolysaccharides
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Membrane Glycoproteins
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Receptors, Cell Surface
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TLR2 protein, human
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Teichoic Acids
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Toll-Like Receptor 2
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Toll-Like Receptors
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Tumor Necrosis Factor-alpha
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Interleukin-10
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lipoteichoic acid
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Interferon-gamma