[Effect and mechanism of arsenic trioxide on chemosensitivity of human lung adenocarcinoma cells]

Zhonghua Jie He He Hu Xi Za Zhi. 2003 Nov;26(11):689-92.
[Article in Chinese]

Abstract

Objective: To study the apoptosis and chemosensitive effect of As(2)O(3) on human lung adenocarcinoma cell line A549.

Methods: MTT colorimetric assay, immunohistochemical method, flow cytometry and RT-PCR were applied to observe the different effects induced by As(2)O(3) with different concentrations on A549 cells.

Results: 1, 2 micro mol/L As(2)O(3) increased Fas expression and inhibited the expression of bcl-2, MRP and LRP; G(1) arrest was observed and the cells were more sensitive to cisplatin. A549 cells in the presence of 5 micro mol/L As(2)O(3) showed growth inhibition and up-regulation of bcl-2, MRP and LRP expressions; but their chemosensitivity to cisplatin did not change.

Conclusion: As(2)O(3) in low concentrations can increase the chemosensitivity of A549 cells by regulating the expression of apoptosis genes, anti-apoptosis genes and drug-resistant genes and affecting the cell cycle progress.

MeSH terms

  • Adenocarcinoma / drug therapy*
  • Adenocarcinoma / pathology
  • Antineoplastic Agents / pharmacology*
  • Apoptosis / drug effects
  • Arsenic Trioxide
  • Arsenicals / pharmacology*
  • Cell Cycle / drug effects
  • Cell Line, Tumor
  • Dose-Response Relationship, Drug
  • Humans
  • Lung Neoplasms / drug therapy*
  • Lung Neoplasms / pathology
  • Multidrug Resistance-Associated Proteins / analysis
  • Neoplasm Proteins / analysis
  • Oxides / pharmacology*
  • Proto-Oncogene Proteins c-bcl-2 / analysis
  • Vault Ribonucleoprotein Particles

Substances

  • Antineoplastic Agents
  • Arsenicals
  • Multidrug Resistance-Associated Proteins
  • Neoplasm Proteins
  • Oxides
  • Proto-Oncogene Proteins c-bcl-2
  • Vault Ribonucleoprotein Particles
  • major vault protein
  • Arsenic Trioxide