The mechanisms of prednisone inhibition of inflammation in Crohn's disease involve changes in intestinal permeability, mucosal TNFalpha production and nuclear factor kappa B expression

Aliment Pharmacol Ther. 2003 Aug 1;18(3):309-17. doi: 10.1046/j.1365-2036.2003.01611.x.

Abstract

Background: The clinical course of Crohn's disease after the induction of remission with medical therapy is characterized by unpredictable relapse.

Aim: To evaluate three surrogate markers, intestinal permeability, mucosal TNFalpha and nuclear factor (NF)-kappaB/IkappaBalpha expression, in order to determine the relationship of these parameters to clinical relapse.

Methods: Thirty patients with active Crohn's disease were treated with a 10 week course of prednisone using a tapering dosing regimen. Intestinal permeability (lactulose/mannitol [L/M ratio]) was determined at baseline and at the end of prednisone tapering. TNFalpha production and the levels of expression of NF-kappaB/IkappaBalpha were measured in colonic mucosal biopsies obtained after the induction of remission.

Results: Twenty-two patients (73%) achieved remission and 50% of patients experienced a clinical relapse during the ensuing 12 months. Treatment with prednisone resulted in a significant decrease in the L/M ratio. Of the patients that relapsed, 75% had a raised L/M ratio at the time of remission compared with 20% of patients with a normal L/M ratio (P < 0.008; hazard ratio = 6.094; CI 1.55, 17.43). Mucosal TNFalpha production was greater in relapsers compared with those who remained in remission. The levels of NF-kappaB in relapsers were significantly greater and levels of cytosolic IkappaBalpha were significantly lower compared with those measured in patients who remained in remission.

Conclusions: These findings underscore the importance of incorporating biological parameters of inflammation in determining the clinical course of Crohn's disease.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Blotting, Western
  • Crohn Disease / drug therapy*
  • Female
  • Humans
  • I-kappa B Proteins / metabolism*
  • Intestinal Mucosa / metabolism
  • Male
  • Middle Aged
  • NF-kappa B / metabolism*
  • Permeability / drug effects
  • Prednisone / pharmacology
  • Prednisone / therapeutic use*
  • Tumor Necrosis Factor-alpha / metabolism*

Substances

  • I-kappa B Proteins
  • NF-kappa B
  • Tumor Necrosis Factor-alpha
  • Prednisone