Abstract
A submicroscopic deletion containing SOX2 was identified at the 3q breakpoint in a child with t(3;11)(q26.3;p11.2) associated with bilateral anophthalmia. Subsequent SOX2 mutation analysis identified de novo truncating mutations of SOX2 in 4 of 35 (11%) individuals with anophthalmia. Both eyes were affected in all cases with an identified mutation.
MeSH terms
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Anophthalmos / genetics*
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Chromosomes, Human, Pair 3*
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Codon, Nonsense
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DNA Mutational Analysis*
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DNA-Binding Proteins / genetics*
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Databases as Topic
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Family Health
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Female
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Gene Deletion
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HMGB Proteins
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Heterozygote
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Humans
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Introns
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Male
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Microphthalmos / genetics
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Models, Genetic
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Molecular Sequence Data
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Mutation
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Nuclear Proteins / genetics*
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Phenotype
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SOXB1 Transcription Factors
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Transcription Factors
Substances
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Codon, Nonsense
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DNA-Binding Proteins
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HMGB Proteins
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Nuclear Proteins
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SOX2 protein, human
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SOXB1 Transcription Factors
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Transcription Factors
Associated data
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GENBANK/AC117415
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GENBANK/AC125613
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GENBANK/AL157425
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GENBANK/AQ473552