Pro12Ala polymorphism in the peroxisome proliferator-activated receptor-gamma (PPAR-gamma) gene and risk of prostate cancer among men in a large cancer prevention study

Cancer Lett. 2003 Feb 28;191(1):67-74. doi: 10.1016/s0304-3835(02)00617-1.

Abstract

The nuclear hormone receptor peroxisome proliferator-activated receptor-gamma (PPAR-gamma) may play a role in prostate carcinogenesis. We examined the association between the PPAR-gamma Pro12Ala polymorphism and prostate cancer risk in a cohort of Finnish male smokers. In a nested case-control analysis that included 193 prostate cancer cases and 188 matched controls, we found no significant association between this polymorphism and prostate cancer risk (odds ratio, OR=1.27, 95% confidence interval, CI: 0.83-1.94), or significant trend or association with tumor stage (OR=1.28, 95% CI: 0.54-3.04 for metastatic disease) or grade (OR=1.57, 95% CI: 0.63-3.91 for poorly differentiated disease). The Pro12Ala polymorphism does not appear to play a significant role in prostate cancer risk in this cohort of men.

Publication types

  • Clinical Trial
  • Randomized Controlled Trial
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adenocarcinoma / epidemiology*
  • Adenocarcinoma / genetics
  • Adenocarcinoma / pathology
  • Adenocarcinoma / prevention & control
  • Aged
  • Amino Acid Substitution*
  • Anticarcinogenic Agents / therapeutic use
  • Case-Control Studies
  • Cell Differentiation
  • Cohort Studies
  • DNA Mutational Analysis
  • Finland / epidemiology
  • Genetic Predisposition to Disease
  • Genotype
  • Humans
  • Incidence
  • Male
  • Middle Aged
  • Odds Ratio
  • Polymorphism, Single Nucleotide*
  • Prostatic Neoplasms / epidemiology*
  • Prostatic Neoplasms / genetics
  • Prostatic Neoplasms / pathology
  • Prostatic Neoplasms / prevention & control
  • Receptors, Cytoplasmic and Nuclear / genetics*
  • Receptors, Cytoplasmic and Nuclear / physiology
  • Risk
  • Smoking / epidemiology
  • Transcription Factors / genetics*
  • Transcription Factors / physiology
  • alpha-Tocopherol / therapeutic use
  • beta Carotene / therapeutic use

Substances

  • Anticarcinogenic Agents
  • Receptors, Cytoplasmic and Nuclear
  • Transcription Factors
  • beta Carotene
  • alpha-Tocopherol