Hematopoiesis-restricted minor histocompatibility antigens HA-1- or HA-2-specific T cells can induce complete remissions of relapsed leukemia

Proc Natl Acad Sci U S A. 2003 Mar 4;100(5):2742-7. doi: 10.1073/pnas.0530192100. Epub 2003 Feb 24.

Abstract

Donor lymphocyte infusion (DLI) into patients with a relapse of their leukemia or multiple myeloma after allogeneic stem cell transplantation (alloSCT) has been shown to be a successful treatment approach. The hematopoiesis-restricted minor histocompatibility antigens (mHAgs) HA-1 or HA-2 expressed on malignant cells of the recipient may serve as target antigens for alloreactive donor T cells. Recently we treated three mHAg HA-1- and/or HA-2-positive patients with a relapse of their disease after alloSCT with DLI from their mHAg HA-1- and/or HA-2-negative donors. Using HLA-A2HA-1 and HA-2 peptide tetrameric complexes we showed the emergence of HA-1- and HA-2-specific CD8(+) T cells in the blood of the recipients 5-7 weeks after DLI. The appearance of these tetramer-positive cells was followed immediately by a complete remission of the disease and restoration of 100% donor chimerism in each of the patients. Furthermore, cloned tetramer-positive T cells isolated during the clinical response specifically recognized HA-1 and HA-2 expressing malignant progenitor cells of the recipient and inhibited the growth of leukemic precursor cells in vitro. Thus, HA-1- and HA-2-specific cytotoxic T lymphocytes emerging in the blood of patients after DLI demonstrate graft-versus-leukemia or myeloma reactivity resulting in a durable remission. This finding implies that in vitro generated HA-1- and HA-2-specific cytotoxic T lymphocytes could be used as adoptive immunotherapy to treat hematological malignancies relapsing after alloSCT.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Bone Marrow Cells / cytology
  • CD8 Antigens / biosynthesis
  • CD8-Positive T-Lymphocytes / metabolism
  • Cell Division
  • Chromium Radioisotopes
  • Chromosomes, Human, X
  • Chromosomes, Human, Y
  • Female
  • Fusion Proteins, bcr-abl / metabolism
  • Genes, MHC Class I
  • Genetic Markers
  • Hematopoiesis*
  • Humans
  • Immunotherapy / methods
  • In Situ Hybridization, Fluorescence
  • Leukemia / drug therapy*
  • Leukemia / pathology*
  • Male
  • Middle Aged
  • Minor Histocompatibility Antigens / pharmacology*
  • Models, Genetic
  • Neoplasm Proteins / pharmacology*
  • Oligopeptides / pharmacology*
  • Peptides / chemistry
  • Phenotype
  • Recurrence
  • Remission Induction
  • Stem Cell Transplantation
  • Time Factors
  • Transplantation, Homologous

Substances

  • CD8 Antigens
  • Chromium Radioisotopes
  • Genetic Markers
  • HA-1 antigen
  • HA-2 antigen
  • Minor Histocompatibility Antigens
  • Neoplasm Proteins
  • Oligopeptides
  • Peptides
  • Fusion Proteins, bcr-abl