Differentiation of geriatric major depression from Alzheimer's disease with CSF tau protein phosphorylated at threonine 231

Am J Psychiatry. 2003 Feb;160(2):376-9. doi: 10.1176/appi.ajp.160.2.376.

Abstract

Objective: Differentiation of geriatric major depression from Alzheimer's disease is hampered by overlapping symptoms. Increased CSF concentrations of tau protein phosphorylated at threonine 231 (p-tau(231)) have been suggested as a biomarker for Alzheimer's disease. The authors asked whether p-tau(231) levels improve the differential diagnosis between geriatric major depression and Alzheimer's disease.

Method: Included were 34 depression subjects, 64 with probable Alzheimer's disease, 17 with possible Alzheimer's disease, and 21 healthy comparison subjects. P-tau(231) concentrations were measured with an enzyme-linked immunosorbent assay.

Results: P-tau(231) levels were significantly higher in Alzheimer's disease than in geriatric major depression patients and healthy comparison subjects. For differentiation of probable Alzheimer's disease from major depression, p-tau(231) correctly allocated 87% of subjects. When possible mild Alzheimer's disease was compared to major depression, p-tau(231) correctly allocated 78% of subjects.

Conclusions: CSF p-tau(231) should be evaluated as a potential biological marker for differentiation of geriatric depression from Alzheimer's disease.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Alzheimer Disease / cerebrospinal fluid
  • Alzheimer Disease / diagnosis*
  • Biomarkers / cerebrospinal fluid
  • Depressive Disorder / cerebrospinal fluid
  • Depressive Disorder / diagnosis*
  • Diagnosis, Differential
  • Enzyme-Linked Immunosorbent Assay
  • Female
  • Geriatric Assessment
  • Humans
  • Male
  • Phosphorylation
  • Threonine / metabolism
  • tau Proteins / cerebrospinal fluid*
  • tau Proteins / metabolism

Substances

  • Biomarkers
  • tau Proteins
  • Threonine