Functional defects in troponin and the systems biology of heart failure

J Mol Cell Cardiol. 2002 Jul;34(7):689-93. doi: 10.1006/jmcc.2002.2028.
No abstract available

Publication types

  • Comment
  • Editorial
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Cardiomyopathy, Dilated / etiology
  • Cardiomyopathy, Dilated / genetics
  • Cardiomyopathy, Dilated / metabolism
  • Cyclic AMP-Dependent Protein Kinases / metabolism
  • Cytoskeletal Proteins / genetics
  • Heart Failure / etiology
  • Heart Failure / metabolism*
  • Humans
  • MAP Kinase Signaling System
  • Mice
  • Phosphorylation
  • Protein Kinase C / metabolism
  • Protein Processing, Post-Translational
  • Sarcomeres / genetics
  • Troponin / genetics
  • Troponin / metabolism*

Substances

  • Cytoskeletal Proteins
  • Troponin
  • Cyclic AMP-Dependent Protein Kinases
  • Protein Kinase C