Abstract
Metabolites of the non-steroidal anti-inflammatory drug Sulindac inhibit cell proliferation by affecting several intracellular signaling pathways including the tumorigenic Ras/Raf/MAPK pathway. Here, we report the synthesis of eight new indene derivatives derived from the Sulindac structure, and present data on their anti-proliferative properties and their effects on the p21ras protein.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Animals
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Anti-Inflammatory Agents, Non-Steroidal
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Antineoplastic Agents / chemical synthesis*
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Antineoplastic Agents / pharmacology
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Cell Division / drug effects
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Combinatorial Chemistry Techniques
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Cyclooxygenase Inhibitors / chemical synthesis
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Cyclooxygenase Inhibitors / pharmacology
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Dogs
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Humans
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Indenes / chemical synthesis
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Indenes / pharmacology*
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Inhibitory Concentration 50
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Mice
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Oncogene Protein p21(ras) / antagonists & inhibitors
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Oncogene Protein p21(ras) / drug effects
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Prostaglandin-Endoperoxide Synthases / drug effects
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Sulindac / analogs & derivatives*
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Tumor Cells, Cultured / drug effects
Substances
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Anti-Inflammatory Agents, Non-Steroidal
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Antineoplastic Agents
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Cyclooxygenase Inhibitors
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Indenes
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Sulindac
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Prostaglandin-Endoperoxide Synthases
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Oncogene Protein p21(ras)