Deactivation and desensitization of mouse embryonic- and adult-type nicotinic receptor channel currents

Neurosci Lett. 2001 Jul 13;307(2):89-92. doi: 10.1016/s0304-3940(01)01929-2.

Abstract

Recombinant nicotinic acetylcholine receptor (nAChR) channels transiently expressed in HEK293 cells were investigated using the patch-clamp technique in the cell-attached and outside-out modes for single-channel analysis and ultra-fast agonist application to multiple channels. Deactivation (current decay after removal of agonist) and desensitization (current decay in the presence of agonist) were analyzed at embryonic- (gamma) and adult-type (epsilon) nAChR channels. Time constants of desensitization were similar for both receptor types (epsilon: 53.1+/-16.9 ms; gamma: 49.2+/-15.7 ms) and corresponded to the mean duration of clusters of single channel openings activated by pulses of 1 mM ACh. Deactivation showed distinct characteristics. Time constants were 1.76+/-0.16 ms for epsilon- and 3.19+/-0.18 ms for gamma-nAChR channels, corresponding to mean burst duration analyzed from single channels in the same preparation (epsilon: 1.85+/-1.2 ms, gamma: 3.85+/-2.1 ms). It is assumed that differences in deactivation are of functional relevance at the muscle endplate.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acetylcholine / pharmacology
  • Age Factors
  • Animals
  • Cells, Cultured / drug effects
  • Cells, Cultured / metabolism
  • Fetus
  • Humans
  • Ion Channels / drug effects*
  • Ion Channels / metabolism
  • Membrane Potentials / drug effects*
  • Membrane Potentials / physiology
  • Mice
  • Neuromuscular Junction / drug effects*
  • Neuromuscular Junction / embryology*
  • Neuromuscular Junction / metabolism
  • Nicotinic Agonists / pharmacology
  • Receptors, Nicotinic / drug effects*
  • Receptors, Nicotinic / genetics
  • Receptors, Nicotinic / metabolism
  • Synaptic Transmission / drug effects*
  • Synaptic Transmission / physiology
  • Transfection

Substances

  • Ion Channels
  • Nicotinic Agonists
  • Receptors, Nicotinic
  • Acetylcholine