Abstract
Sumatriptan, a h5-HT1D and h5-HT1B receptor agonist used clinically as a migraine-abortive, produces certain side effects thought to result from its affinity for h5-HT1B receptors. The present investigation extends our work with benzylimidazolines as novel non-tryptamine h5-HT(1D/1B) ligands. The effect of N-methylation, N-benzylation, ring-aromatization, and variation of the imidazoline ring on affinity both at h5-HT1D and h5-HT1B receptors was examined. Several compounds were identified with good affinity and enhanced (i.e., > 100-fold) h5-HT1D versus hS-HT1B selectivity.
MeSH terms
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Benzyl Compounds / chemical synthesis
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Benzyl Compounds / metabolism
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Benzyl Compounds / pharmacology*
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Cell Membrane / chemistry
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Cell Membrane / metabolism
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Humans
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Imidazoles / chemical synthesis
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Imidazoles / metabolism
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Imidazoles / pharmacology*
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Ligands
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Migraine Disorders / drug therapy
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Protein Binding
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Radioligand Assay
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Receptor, Serotonin, 5-HT1B
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Receptor, Serotonin, 5-HT1D
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Receptors, Serotonin / metabolism*
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Serotonin Agents / chemical synthesis*
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Serotonin Agents / metabolism
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Serotonin Agents / pharmacology
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Serotonin Receptor Agonists / pharmacology*
Substances
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Benzyl Compounds
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HTR1B protein, human
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Imidazoles
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Ligands
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Receptor, Serotonin, 5-HT1B
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Receptor, Serotonin, 5-HT1D
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Receptors, Serotonin
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Serotonin Agents
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Serotonin Receptor Agonists