Cutting edge: IL-15 costimulates the generalized Shwartzman reaction and innate immune IFN-gamma production in vivo

J Immunol. 2000 Feb 15;164(4):1643-7. doi: 10.4049/jimmunol.164.4.1643.

Abstract

Sequential administration of LPS to SCID mice results in the generalized Shwartzman reaction, manifesting as rapid mortality via cytokine-induced shock. Here we demonstrate that in vivo neutralization of IL-15 before LPS priming significantly reduced lethality in this reaction (p = 0.0172). We hypothesize that LPS priming induces IL-12 and IL-15 that costimulate NK cell-derived IFN-gamma. Such IFN-gamma may then in turn sensitize macrophages to elicit the Shwartzman reaction following a subsequent LPS challenge. Supporting this, IL-12 and IL-15 synergized to induce murine NK cell IFN-gamma production in vitro. LPS stimulation of SCID mouse splenocytes resulted in measurable IFN-gamma production, which was reduced when IL-15 was neutralized or IL-2/15Rbeta was blocked. Pretreatment with either anti-IL-2/15Rbeta or anti-IL-15 Abs reduced serum IFN-gamma protein following LPS administration to SCID mice. Collectively, these data provide the first in vivo evidence that IL-15 participates in LPS-induced innate immune IFN-gamma production and significantly contributes to the lethal Shwartzman reaction.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Female
  • Immune Sera / pharmacology
  • Immunity, Innate
  • Injections, Intravenous
  • Interferon-gamma / antagonists & inhibitors
  • Interferon-gamma / biosynthesis*
  • Interleukin-15 / antagonists & inhibitors
  • Interleukin-15 / biosynthesis
  • Interleukin-15 / genetics
  • Interleukin-15 / physiology*
  • Lipopolysaccharides / antagonists & inhibitors
  • Lipopolysaccharides / toxicity
  • Mice
  • Mice, SCID
  • RNA, Messenger / biosynthesis
  • Receptors, Interleukin-15
  • Receptors, Interleukin-2 / antagonists & inhibitors
  • Shwartzman Phenomenon / immunology*
  • Shwartzman Phenomenon / mortality
  • Spleen / cytology
  • Spleen / immunology
  • Spleen / metabolism

Substances

  • Il15ra protein, mouse
  • Immune Sera
  • Interleukin-15
  • Lipopolysaccharides
  • RNA, Messenger
  • Receptors, Interleukin-15
  • Receptors, Interleukin-2
  • Interferon-gamma