Another mechanism causing the bystander effect besides the gap junction's role during the glioma gene therapy with HSV-TK/GCV system

Adv Exp Med Biol. 1998:451:133-8. doi: 10.1007/978-1-4615-5357-1_21.
No abstract available

MeSH terms

  • Antiviral Agents / therapeutic use*
  • Cell Division / drug effects
  • Ganciclovir / therapeutic use*
  • Gap Junctions / physiology*
  • Genetic Therapy / methods*
  • Glioma / pathology*
  • Glioma / therapy*
  • Humans
  • Simplexvirus / enzymology*
  • Simplexvirus / genetics
  • Thymidine Kinase / genetics*
  • Thymidine Kinase / metabolism
  • Tumor Cells, Cultured

Substances

  • Antiviral Agents
  • Thymidine Kinase
  • Ganciclovir